Announcing new causes of Alzheimer's disease
New US scientists have discovered that when brain cells are aging and the amount of DNA produced by these cells is not good, they will not divide and start producing harmful proteins.
New scientists claim that the culprit in Alzheimer's disease may be the body's natural defense mechanism for cancer, destroying the brain when we become old.
New US scientists have discovered that when brain cells are aging and the amount of DNA produced by these cells is not good, they will not divide and start producing harmful proteins.
Scientists believe that the immune system, which protects against faulty cells, can develop into tumors, tumors, have been "successful" in protecting the body's internal organs, preventing cancer .
But now, people's lifespan is being enhanced, so the cell conversion process (aging cell process) can be the cause of Alzheimer's disease. As our brain ages, millions of cells also begin to age, which means there won't be enough healthy cells to push plaque (aging cells) that are building up in the brain. of Alzheimer's patients. Plaque causes many cells to stop dividing, causing the brain to age more quickly.
As our brain ages, millions of cells also begin to age, which means there won't be enough healthy cells to push plaque (aging cells) that are building up in the brain. of Alzheimer's patients.
Dr. Claudio Torres of the Philadelphia College of Medicine, Drexel University, put brain cells into H 2 0 2 (hydrogen peroxide) to accelerate the aging of brain cells. He found that brain cells stopped dividing and began producing a large number of proteins prone to inflammation.
Research by Dr. Claudio Torres has shown that in people aged 80 and 90 healthy, up to 30% of brain cells have aging, but in people with Alzheimer's disease, this number is 40%.
He said that his research could open a new direction for the disease. According to Dr. Torres, blocking the aging process of cells can lead to cancer, but eliminating faulty cells can help prevent Alzheimer's disease.
Reference: Telegraph
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