Steel people do not know if pain is due to a genetic mutation

The study published in the latest issue of 'Nature, Genetics' in the UK shows that some cases right from birth did not have pain and common reactions to pain to avoid danger.

The latest report by an international team of scientists shows that the cause of these conditions is gene mutation.

According to scientists, the main cause of congenital pain sensory disease (HSAN) is that in the process of embryonic growth, the outer embryo layer is not developed completely.

The disease is usually classified into 5 types of Type I, Type II, Type III, Type IV and Type V.

Scientists have studied the analysis of members of a family with Type II congenital pain sensory disease, the results showed that the FAM134B gene is located on chromosome 5 of many afflicted members. mutation.

Scientists believe that the FAM134B gene is often found in dorsal root ganglion (DRG), in which neurons are responsible for transmitting primary sensory neurons to the central nervous system.

The discovery study, due to inhibition of FAM134B gene, has led to some dead-rooted neural lymph nodes. This led to the mutation FAM134B gene also inhibited, and led to the neural root ganglia also died, hindering the feeling of human pain, leading to disease of feeling of pain.