Does sugar make us older?
Recently, experiments conducted on mice by biochemistry professor at Montreal University Louis Rokeach have shown that reducing calorie intake will prolong the life of this animal. The phenomenon is explained by the ability of cells to detect sugar in their environment.
Sugar appears in our meals in many different forms of food.(micapatissier.wifeo.com)
Aging is a complex phenomenon that we still cannot understand fully. Aging is very natural, it arises from residues in body metabolism and by regulating gene expression, often accompanied by manifestations such as reducing resistance to stress, losing internal balance. and increased risk of disease.
However, this explanation became increasingly unconvincing. Eating habits, especially high calories, are the cause of our longevity. For example, for rats often fed with glucose syrup, when halving the amount of calories in food, their lifespan will increase to 40%.
The intake of calories into the body depends on two factors: food preferences and digestion. When the amount of assimilated food that is directly delivered to the cells appears the following process: First, the 'sensor' on the cell surface will detect glucose. After that, glucose penetrates into cells and converts into energy.
When studying cell enzymes, researchers at the University of Montreal found that their lifespan would increase if glucose levels were reduced. Continuing to study, they discovered an unexpected phenomenon: when the glucose detector was removed from the cell enzymes, these cell enzymes would have a long lifespan equivalent to the yeast cells. cells do not get much glucose. In other words, cell life depends on the ability to detect glucose in your environment.
On the other hand, the removal of 'sensors' that detect glucose levels increases the lifespan of cells. 'This study allows us to understand the relationship between aging-related diseases and the excessive consumption of sugar in today's diet. The study also opens up new treatments for treating aging-related diseases , 'said Professor Louis Rokeach, lead author of the new study in PLoS Genetics.
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